Research, Articles & Case Studies

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December 1st, 2016

CRANIAL SEA

Kenneth R. Koles, PhD, DSc, RAc, LMT
Curriculums:

Ken Koles talks about what Craniosacral Therapy is and the benefits of this work. 

November 28th, 2016

Scientists discover neuron-producing stem cells in the membranes covering the brain

VIB - Flanders Institute for Biotechnology
Curriculums: Upledger's CranioSacral TestimonialCategory / SomatoEmotional Release, Barral's Visceral Manipulation / Neural Manipulation / New Manual Articular Approach / Manual Approach to the Brain,

​This article is about how unexpected cells have been found in the protective membranes that enclose the brain, the so called meninges. These 'neural progenitors' -- or stem cells that differentiate into different kinds of neurons-- are produced during embryonic development. These findings show that the neural progenitors found in the meninges produce new neurons after birth-- highlighting the importance of meningeal tissue as well as these cells' potential in the development of new therapies for brain damage and neurodegeneration. A paper highlighting the results was published in the leading scientific journal Cell Stem Cell.

November 24th, 2016

Manual Therapies Reduce Pain Associated with Trigeminal Neuralgia

Susan Vaughan Kratz
Curriculums:

Abstract 

Introduction: Trigeminal Neuralgia {TN) can be an extremely debilitating condition effecting quality of life, emotional well-being, and engagement in daily occupation. Surgical and medication treatments are cited extensively through the literature but can have undesired side effects, can lose effectiveness over time, or are quite invasive. Little is reported about craniosacral therapy, lymphatic drainage, or other gentle manual techniques as treatment options. 

Objective: This paper introduces and summarizes the experiential process and outcomes of three adults receiving manual therapies to treat TN. This review investigates low-risk, conservation clinical options and explores for treatment guidelines for TN. 

Method: Chart review and client interviews in multiple follow-up contacts of a convenience sample to explore immediate and long term outcomes. All treatment techniques utilized per clients are summarized, and include: Upledger's CranioSacral Therapy (U-CST); Chikly's brain curriculum for lymphatic enhancement and nerve down-regulation techniques; and Wanveer's glia structure and glymphatic mobilization techniques. One occurrence of a spontaneous Somato-Emotional Release technique was also called for. Measurement of baseline and outcomes was conducted using: Verbal descriptor scale; Verbal numeric rating scale; Visual analog scale (VAS); and Self-report of quality of life and pain's impact upon performing daily activities. Data were analyzed using qualitative content analysis 

Conclusion: Three adults reported individual positive changes or resolution of TN pain. All three reported restoration of quality of life and emotional well-being. One made use of techniques for self-help Comparison to other methods, or variations of these methods utilizing the same names or terminologies, should be avoided. This report is an attempt to aid in the needed clarification between different approaches used in clinical practices. Positive responses suggest that these methods hold value for further study as a viable treatment option to address the agony of neuralgia. 

November 18th, 2016

The sacral autonomic outflow is sympathetic

I. Espinosa-Medina O. Saha, F. Boismorea
Curriculums:

A kinship between cranial and pelvic visceral nerves of vertebrates has been accepted for a century. Accordingly, sacral preganglionic neurons are considered parasympathetic, as are their targets in the pelvic ganglia that prominently control rectal, bladder, and genital functions. Here, we uncover 15 phenotypic and ontogenetic features that distinguish pre- and postganglionic neurons of the cranial parasympathetic outflow from those of the thoracolumbar sympathetic outflow in mice. By every single one, the sacral outflow is indistinguishable from the thoracolumbar outflow. Thus, the parasympathetic nervous system receives input from cranial nerves exclusively and the sympathetic nervous system from spinal nerves, thoracic to sacral inclusively. This simplified, bipartite architecture offers a new framework to understand pelvic neurophysiology as well as development and evolution of the autonomic nervous system.
November 16th, 2016

Having my head rubbed like a baby cured my stress: After her father died and she lost her job at a glossy magazine all in the same year, one woman tries craniosacral therapy

Niki Browes
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Niki Browes writes in the The Daily Mail News about her continual low-level anxiety — which manifested in tense head and neck aches even painkillers couldn’t help — that made her decide something needed to be done to make her to feel like herself again. She said “A friend mentioned she’d been having craniosacral therapy — George Clooney was an advocate! — and found it restorative, even healing.” This is her story.
November 13th, 2016

Study reveals role of spleen in prolonged anxiety after stress

Provided by The Ohio State University
Curriculums: Upledger's CranioSacral TestimonialCategory / SomatoEmotional Release, Barral's Visceral Manipulation / Neural Manipulation / New Manual Articular Approach / Manual Approach to the Brain,

In this study, the trio of scientists determined that the immune cell changes persisted for almost a month after the mice experienced the stress.

November 11th, 2016

What Veterans Want You To Know About PTSD

Carolyn Gregoire
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Here are five things vets wish others knew about PTSD.
November 10th, 2016

Pain is not just a matter of nerves

Medical University of Vienna
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The sensation of pain occurs when neural pathways conduct excitation generated by tissue damage to the spinal cord, where the nociceptive information is extensively preprocessed. From there, the information is transmitted to the human brain, where the sensation of "pain" is finally created. This is the general belief. However, researchers have now discovered that pain is not just a matter of nerves but that non-neuronal cells, the glial cells, are also involved in clinically relevant pain models and their activation is sufficient to amplify pain.
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